Jeremy T. Hetzel
A native of Philadelphia, Jeremy came to the Chicagoland in 2002 to study philosophy and biology at the University of Chicago. He began his collaboration with Dr. Rubin in the fall of 2004 and became a full-time research assistant upon graduation from the College in spring of 2006.
Jeremy's research interests are currently broad, ranging from genetic mechanisms of disease and predictors of disease activity to novel imaging techniques and medical ethics. A particular endeavor of his is to better understand how biological, psychological, and social factors interact to effect disease activity and overall patient quality of life, especially in the inflammatory bowel diseases. While psycho-social factors such as stress are often cited as possible causes in disease etiology, current study methodologies are generally inadequate at collecting empirical evidence to study how psycho-social factors correlate with biological disease activity. Jeremy hopes that multi-disciplinary collaboration will lead to a better understanding these complex interactions and improve patient quality of life.
Jeremy's research interests are currently broad, ranging from genetic mechanisms of disease and predictors of disease activity to novel imaging techniques and medical ethics. A particular endeavor of his is to better understand how biological, psychological, and social factors interact to effect disease activity and overall patient quality of life, especially in the inflammatory bowel diseases. While psycho-social factors such as stress are often cited as possible causes in disease etiology, current study methodologies are generally inadequate at collecting empirical evidence to study how psycho-social factors correlate with biological disease activity. Jeremy hopes that multi-disciplinary collaboration will lead to a better understanding these complex interactions and improve patient quality of life.
Recent publications:
- Puri N, A Khramtsov, S Ahmed, V Nallasura, JT Hetzel, R Jagadeeswaran,G Karczmar, R Salgia. A selective small molecule inhibitor of c-Met, PHA665752, inhibits tumorigenicity and angiogenesis in mouse lung cancer xenografts. Cancer Research. 2007;67(8):3529-34.
- Rubin DT, JA Rothe, JT Hetzel, RD Cohen, SB Hanauer. Are dysplasia and colorectal cancer endoscopically visible in patients with ulcerative colitis? Gastrointest Endosc. 2007;65(7):998-1004.
